Fig. 7From: Antiviral mechanism of carvacrol on HSV-2 infectivity through inhibition of RIP3-mediated programmed cell necrosis pathway and ubiquitin-proteasome system in BSC-1 cellsEffect of HSV-2 infection and carvacrol treatment on the RIP3 programmed cell necrosis pathway and the ubiquitin-proteasome degradation pathway on infected cells. a the RIP3 programmed cell necrosis pathway; b the ubiquitin-proteasome system. Red arrow represent: effect of HSV-2 infection on cells; Blue arrow represent: effect of carvacrol treatment on HSV-2 infected cellsBack to article page