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Fig. 4 | BMC Infectious Diseases

Fig. 4

From: Antiviral mechanism of carvacrol on HSV-2 infectivity through inhibition of RIP3-mediated programmed cell necrosis pathway and ubiquitin-proteasome system in BSC-1 cells

Fig. 4

Immunofluorescence staining results of gB and RIP3 proteins in normal and infected cells. a, b, c: the normal cells; d, e, f: the infected cells. g-p:The gene and protein expression levels of RIP3, TNF-α, and MLKL in cells treated with the different post-infection time of HSV-2 and different carvacrol concentrations

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BMC Infectious Diseases

ISSN: 1471-2334

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