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Fig. 1 | BMC Infectious Diseases

Fig. 1

From: Antiviral mechanism of carvacrol on HSV-2 infectivity through inhibition of RIP3-mediated programmed cell necrosis pathway and ubiquitin-proteasome system in BSC-1 cells

Fig. 1

The viral titer determination of HSV-1 and HSV-2 on different cell lines. a Vero cells infected with HSV-1 at 10− 3 dilution; (b) BSC-1 cells infected with HSV-1 at 10− 3 dilution; (c) Vero cells infected with HSV-2 at 10− 6 dilution; (d) BSC-1 cells infected with HSV-2 at 10− 6 dilution. The morphological changes of BSC-1 cells infected HSV-1 or HSV-2 (200 ×). (e) The normal BSC-1 cells; (f) BSC-1 cells infected with HSV-1 for 24 h; (g) BSC-1 cells infected with HSV-2 for 24 h; (h) BSC-1 cells infected with HSV-2 for 30 h. The cellular survival curves of BSC-1 cells treated with carvacrol and 2% true carvacrol solution. i carvacrol; (j) 2% true carvacrol solution. The antiviral activities of carvacrol against HSV-1/HSV-2. (K) HSV-1; (I) HSV-2. *, p < 0.05; **, p < 0.01; ***, p < 0.001

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