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Table 4 Stepwise logistic regression analysis of factors associated with the development of leprosy and its clinical forms

From: Influence of KIR genes and their HLA ligands in the pathogenesis of leprosy in a hyperendemic population of Rondonópolis, Southern Brazil

Variable response Variable independent Coefficient (β) P OR -2Log
per se vs. controls KIR2DL1 -2.227 0.001 0.10 1060.2
KIR2DL1 - C2/C2 0.431 0.022 1.54
KIR2DS2 - C1 0.537 0.002 1.71
LL vs. controls Gender -1.217 0.047 0.29 141.9
KIR2DL1 -2.815 0.001 0.06
KIR2DL2/2DL2 - C1 1.612 0.042 5.01
Borderline vs. controls KIR2DL1 -1.868 0.001 0.15 936.9
TT vs. controls Gender 0.755 0.017 2.13 288.5
Ethnic 0.365 0.048 1.44
KIR2DL1 e -3.708 0.001 0.02
KIR2DL1 - C2/C2 1.612 0.001 5.01
KIR2DL2 - C1 1.452 0.001 4.27
KIR2DL2/2DL2 - C1 1.173 0.059 3.23
TT vs. Borderline Gendera 0.947 0.002 2.57 281.7
Ethnic 0.410 0.017 1.50
KIR2DL1 -1.809 0.001 0.16
KIR2DL1 - C2/C2 1.932 0.001 6.90
KIR2DL2 - C1 1.840 0.001 6.30
TT vs. LL Gender 1.783 0.004 5.95 82.4
Borderline vs. LL KIR2DL2/2DL2 - C1 -1.579 0.024 0.206 151.3
  1. NOTE: To this analysis, only KIR genes that were significant in the chi-square analysis were considered. (β): Coefficient of variables; -2Log: Logarithm of chance. Tuberculoid (TT), Borderline (BL, BB, BT), and Lepromatous (LL).